A second massive fish kill being caused by the same Moreton Bay Council's KBR works.
REPORT TO COUNCIL for the Beachmere Committee - 3-2-2010 -
For the second time the Moreton Bay Beachmere lake is falling victim to a massive FISH KILL and again the Council, and it's commissioned fish experts KBR are telling Media a box of LIES; Its only 72 days since the last two tons of fish died in this lake because of AMMONIA poisons from the Council's KBR works.
We tested the water seeping out of the same KBR Council's works area and found it to have such a high level of Ammonia, it was off the scale. Unfortunately, through successive heavy rains falls over the last week or so, this toxin was repeatedly washed into the lake again.
Two things occurred; 1; It triggered an ALGAE BLOOM being fed by effluent from all the area, and 2, The water became Loaded with Ammonia which is the PRIME cause of the Fish-kill.
Council rigged up air compressors again to feed air into the lake, and as last time; The fish were not congregating anywhere near these turbulent waters looking for Oxygen, but avoided this oxygen saturated area and were trying to get out of the lake, showing stress and dying of the poison.
It seems there's little or NO integrity or Honesty left in the Governments. Myself and two other experts in the maintenance of MARINE LIFE gave this Council reports that PROVED the fish kill was as a result of water directly from the 'SEWERAGE WORKS' However; they dismissed our reports, and chose to accept a fraudulent false report from KBR which was worded to prop up their actions and to avoid liability. We objected to this report, and we totally rejected it at the full Council meeting that we attended and we accused the Council of trying to 'white-wash the issue.' AT this same Council meeting, the KBR representative Doug Shooter refused to allow us to test samples of the water for ammonia at the table. We wanted to show the whole meeting the ammonia test results.
TV NEWS CHANNEL 9, 10, took complete films of the source of the poisoned water, BUT CUT THE FILM report, and did not show the truth of what happened. Obviously this was to protect the Mayor Allan Sutherland, KBR, and the EPA Scientist Munroe Mortimer from public backlash.
We ask - "How can we have confidence in the Media when they BLATANTLY withhold evidence and BLATANTLY allow Politicians and Government employees to hide behind LIES.
We invite both Mayor Allan Sutherland and Scientist from EPA Munroe Mortimer to answer our written questions tabled at the last meeting, AND we offer copies of these documents to any interested people in the area.
This film shows the whole area and the source of the pollution. BUT still the Council and KBR have their men running up and down the lake in boat taking water samples at different depths no doubt to come up with another bogus report on the issues.
Its amazing, BUT we didn't see any one actually taking samples of the actual black sludge water entering into the lake. We were there for about 5 hours but didn't see anyone including the lady from the EPA take any testing of this stinking polluted water. She just took some pictures.
We now wait for another EPA report to 'white-wash' the Council's management. Our guess is: "They will again put blame on the 'Lack of Oxygen, and the Algae bloom?" that is causing the fish kill. If they do this; It will again be a pack of LIES and again it will show that these people both from Moreton Bay Council and the EPA are not to be trusted.
John Howard was with Mr Dave Lesar, Chairman, President and CEO of Halliburton at the opening of The Ghan railway in 2004. Halliburton put up 50% of the money to build the Ghan railway (without any tendering process) which now transports uranium from South Australia to Darwin. Halliburton is the company Dick Cheney was CEO of before becoming Vice-President of the United States – and the company that still pays him up to $1million a year in deferred compensation. In his article Australia: the new 51st state published in the New Statesman in March this year, John Pilger writes that Howard’s servility to the US is even greater than Tony Blair’s and has earned him the nickname Bush’s deputy sheriff.
Why are these key people of the NWO - US administration and International crime syndicate allowed to take over our 'Water' Electricity' 'Transport' Roads' Rails' etc, and actually buy up all of our public owned assets.?
(Recently) ” Vice President Dick Cheney came to Sydney to “thank” Howard for his support. The New South Wales state government rushed through a law that allowed Cheney’s 70 secret service guards to carry live weapons. With the police, they took over the centre of Sydney and closed the Harbour Bridge and much of the historic Rocks area. Seventeen-vehicle motorcades swept theatrically here and there, as if Howard was boasting to Cheney: “Look at my control over this society; look at my compliant country.” And yet his guest and mentor is a man who, having refused to fight in Vietnam, has brought back torture and lied incessantly about Iraq, who has made millions in stock options as his Halliburton company profits from the carnage and who has vetoed peace with Iran. Almost every speech he gives includes a threat. By any measure of international law, Cheney is a major war criminal.”(Member of the CFR And leader of the International crime and banking syndicate)
Halliburton is now under investigation by the US Nuclear Regulatory Commission however its new railway line between Alice Springs and Darwin (constructed by its subsidiary Kellogg, Brown and Root KBR) passes conveniently closely to all three sites commonly expected to become nuclear waste disposal sites, and would be an ideal way to transport nuclear spent fuel arriving either in Darwin, Port Adelaide, or any eastern Australian seaboard port.
REPORT TO Nov 2009
Mayor Allan Sutherland Moreton Bay Council
and the Beachmere Lake Committee. Copy to Media.
Further to our conversations and my presentation to your Council meeting on Friday 27th regarding the Beachmere Lake fish kill;
1. I make it very clear; the prime cause of the fish kill is AMMONIA toxin sourced through the pumping effluent of substrate water and releasing it back into the lake. And all of your employees should be made aware of this blunder so it will never happen again. It was obvious to me at your meeting that those academics present at the meeting last Friday 27th Nov, who were responsible for allocating the contractors to go into the area and to pump out 'substrate' water and to release this poison back into the pristine lake were completely oblivious of the dangers in issuing such orders to sink deep holes to accommodate the sewerage overflow tanks. These persons may have "book-knowledge' of engineering but have NO PRACTICAL EXPERIENCE in evaluating what is needed to preserve healthy marine life. Over 5 weeks ago the Council was warned that the inlets-outlets were blocked preventing the tidal flow which resulted in this lake being a "closed marine system' and the Council took no action to rectify the problem.
One gentleman 'Munro' from EPA at the Council meeting tried to water down the findings that this fish-kill was caused by 'Ammonia Nitrate and Ammonia Nitrite, But this film report and the COMPREHENSIVE REPORT FROM BEN DIGGLES shows conclusively that it was 'ammonia' spewed into the lake from the 'substrate' by the actions of the Council contractors that killed all the fish.
2. Hot days and Oxygen were not the prime cause of this eco system's destruction, but oxygen became depleted owing to decomposing fish and detritus matter, and the fact that the lake had become a "Closed system" from lack of Council maintenance to keep the tidal flow operational. Fish that have been traumatised by ammonia toxin generally die because their gill tissue is damaged and they no longer have the ability to absorb oxygen. Most of the fish will die and forcing air into the lake now will have little effect as it's too late.
3. OTHER POTENT CAUSES OF FISH KILLS IS MONSANTO GLYPHOSATE "Roundup" that has been indiscriminately sprayed in gutters and catchment areas by council contractors, and farmers resulting in the poison being washed into the river and area creeks by rain, then the fish kill will emerge some hours later after the rain. It is Glyphosate that has caused the destruction of the marine life and the bleaching of coral along the Queensland Coast. It is Glyphosate that has caused the destruction of the 'aquatic plant-life' in MORETON BAY and the subsequent loss from the area of many Dugong and loss of healthy environment to sustain prawns and crustaceans. Glyphosate is responsible for killing out algae spore which is also part of the initial food chain needed to sustain the newly hatched fish fry.
With the main spawning habitats destroyed; Fish populations in Moreton Bay are now severely reduced as a consequence of this use of Glyphosate.
Remedy: - BAN THE USE OF 'ROUNDUP' and chemical effluent into Moreton Bay and the previous massive fish population and the Dugong will return.
4.. The remedy of the Beachmere lake is to pump in water and to 'flush' out the lake immediately and ensure a proper tidal circulation flow is reintroduced. Otherwise install a fountain in the centre to circulate the water.
5. The blocking off of the water from the adjacent breeding habitat of the swamp mangrove area is blatant ecosystem vandalism and is responsible for the enormous loss of fish stocks in the region, if a private corp did this, there would be enormous retaliation by EPA and millions $$$$ fines imposed.
All interested members of the public, and relevant authorities to assist them in their investigations
Cover: Western shore of the lake at 7 am on 25 Nov 2009, notable by more dead fish and evidence of a recently reduced lake level, even after pumping of over 77 tonnes of groundwater the previous night. Summary
This report details observations and measurements taken by Dr Ben Diggles during an informal inspection of an acute fish kill at Biggs Ave Lake, Beachmere on the evening of 24 Nov 2009 and the early morning of 25 Nov 2009. I present my observations, a brief analysis, and my professional opinion regarding the most plausible reasons for the fish kill event. This report is made in the context of a concerned citizen reporting a first hand account of an incident in an area of investigation which he has significant formal training and experience, in order to assist authorities to determine the cause and prevent repeat events.
To summarise the results of this report, the epidemiology of the fish kill, together with the properties and volumes of the effluent groundwater pumped into the lake, strongly suggests the fish died from toxic exposure to hydrogen sulphide and/or ammonia (possibly complicated by reduced oxygen levels), all of which resulted from a chain of events following pumping of large quantities of groundwater into the lake from the nearby construction site.
Certainly it is the responsibility of government authorities to conduct a thorough investigation into this fish kill, particularly to rule out other (albeit much less likely) potential causes such as pesticide and heavy metal contamination, algal blooms and parasitic infection. However, it is important to point out that the volume of effluent groundwater pumped into the lake alone, in the absence of any other insults, was sufficient to explain:
1. Some or most of the observed reductions in oxygen levels in the lake,
2. The deaths of the fishes in it through toxic exposure, and
3. The observed behavior of the surviving fishes.
After hearing of a large “natural” fish kill in my local area from various forms of media and receiving a few phone calls from interested friends and colleagues who suggested the kill site was worth looking at, I arrived at a lake behind Biggs Ave, Beachmere (Figure 1) at 4.30 pm on Tuesday 24 November 2009 in time to chat to council workers who were running two large aerators (Figure 2). They commented on the large number of dead fish they had just taken away and dumped in a local landfill. Mobile phone photographs were shown of milkfish up to 1 meter long, large mangrove jack (Figure 4) and many mullet, bream, herring and silver biddy (Figure 5). My inspection of the lake margins found large numbers of dead fish as well as a few moribund fish (mainly mullet and yellowfin bream) displaying abnormal behaviour such as slow swimming near the lake edges with their mouths out of the water. Several large mudcrabs were also observed on the edges of the lake in water less than 10 cm deep. At the southern end of the lake (Site 1) there was a construction site (Figure 3) where effluent groundwater was being pumped from a well through a pipe into the lake at a rate (measured with a bucket and stopwatch) of 8 L per 4 seconds (2L/sec). A pungent smell of hydrogen sulphide was being emitted from the water coming from the pipe, and large number of dead fish were observed immediately to the west, blown there by the prevailing E/NE wind (Figure 1). After speaking with local residents on the history of events leading up to the kill I took a water sample from near site B (Figure 1) and returned home to examine it under the light microscope, only to observe a few algal cells and ciliates. As the circumstances of the kill appeared suspicious, and in my professional opinion unlikely to be a result of a natural event, I returned 90 minutes later with more equipment to conduct some basic water testing and further observations of dead and dying fish at a time when the kill was still occurring.
I present my observations, a brief analysis, and my professional opinion regarding the most plausible reasons for the fish kill event. This report is made in the context of a concerned citizen reporting a first hand account of an incident in an area of investigation which he has significant formal training and experience, in order to assist authorities to determine the cause and prevent repeat events.
The levels of ammonia, nitrite, nitrate and pH of water samples from sites 1and B were measured using a colorimetric API Aquarium Pharmaceuticals Master Test Kit as per the manufacturers instructions.
The size of the fish affected by the kill, particularly the mangrove jack and giant herring, suggests that it is an unprecedented event for at least 6 to 8 years. Indeed, local residents cannot recall a fish kill of this magnitude in the area for over 25 years. No significant rainfall events preceded the kill for around two weeks previous, and while the weather was warm during that time it was not unusually warm and similar hot days had preceded the event for several weeks.
Ammonia in groundwater Colorimetric test kits indicated that the groundwater pumped from the construction site held high levels of ammonia as well as the hydrogen sulphide. In fact, in order to get an accurate reading with the colorimetric test kits (which only read up to 8 mg/L), I needed to do a 50:50 dilution with tap water (0
11 – ammonia reading) to bring the samples back into the testable range of the kit (Figure 7).
The maximum reading of the kit was 8 mg/L, and the 50% diluted samples read between 4 and 8 mg/L on the colorimetric scale, suggesting the groundwater had around 10-12 mg/L ammonia content (Figure 7)
The un-ionised form of ammonia is most toxic to marine life (DEH 1998), and it is the un-ionised form that is predominant at higher pH over 7.5 (Eddy et al. 2005). Hence the pH in the lake was high enough to promote ammonia toxicity. During ammonia exposures, whether chronic or episodic, estuarine fish are most at risk of ammonia toxicity at elevated temperatures (Eddy et al. 2005). They are also likely to be at risk from ammonia intoxication in waters of low salinity, high pH and high ammonia levels, as these conditions promote ammonia transfer from the environment into the fish, both as ionized and unionized ammonia, as well as promoting ammonia retention by the fish (Eddy et al. 2005). Clearly conditions in the lake at the time of the kill were suitable for promotion of ammonia toxicity to the fish.
Hydrogen sulphide in groundwater The pungent smell of hydrogen sulphide (H2S) (which was reported by local residents for several weeks before the kill), was very prominent if one stood near the groundwater effluent pipe, and it was clear that the effluent groundwater contained some level of H2S. H 2S is particularly common in groundwater as it is produced by bacteria in oxygen depleted (anoxic) conditions, such as those commonly found in mangrove muds (ANZECC 2000).
H2S is very toxic to fish, with recommended levels in water used for aquaculture being no more than 0.001 to 0.002 mg/L, with detection of any level of H2S considered highly undesirable (ANZECC 2000, Appendix 1,2,3). The toxicity of H2S also tends to increase with increasing water temperature and decreasing dissolved oxygen (Aldelman et al. 1972, Yusoff et al. 1998). The ANZECC (2000) guidelines for protection of aquatic ecosystems suggest the allowable levels of H2S are 900 times less than for ammonia, such is the toxicity of H2S. Without data on the actual hydrogen sulphide content of the groundwater at this time (complex laboratory testing is needed to measure it), it is difficult to ascertain the extent of the role it played in the kill. However the epidemiology of the kill event fits in well with the pathophysiology of H2S toxicity (R. Chong, personal communication). At lower levels of H2S intoxication, the oxygen binding capability of the fishes blood is impaired, and the fish respond by moving to the water surface and gasping for air. However with increasing H2S this toxin suppresses central nervous system activity leading to sudden death without apparent clinical symptoms.
Fish that die from H2S intoxication do not exhibit agonal gasp, which explains the post mortem appearance of the vast majority of fish that died.
Certainly it is the Governments responsibility to conduct a thorough independent investigation into this fish kill, to rule out other (albeit much less likely) potential causes such as pesticide and heavy metal contamination and parasitic infection. However, it is clear that the volume of effluent groundwater pumped into the lake alone was sufficient to explain not only the observed reductions in oxygen levels in the lake, but also the deaths of the fishes in it from ammonia and/or H2S toxicity.
In summary, the epidemiology of the fish kill, together with the properties and volumes of the effluent groundwater pumped into the lake, strongly suggests the fish died from toxic exposure to hydrogen sulphide and/or ammonia, (possibly complicated by reduced oxygen levels) resulting from a chain of events following pumping of large quantities of groundwater into the lake from the nearby construction site.
GOLD COAST BROADWATER HAS ALSO LOST THE MASSIVE FISH AND CRUSTAECEAN POPULATIONS OWING TO COUNCIL'S USE OF GLYPHOSATE.
When the Gold Coast Broadwater lost all the 'sea-grass' and subsequent all the Dugong, the Dugong moved up to the Moreton Bay area only to find that their habitat in this region was destroyed in the same manner.
dead fish report - http://www.youtube.com/watch?v=jePQDh...
Council was warned about this marine habitat vandalism more than 4 weeks ago.